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Gruppiert nach: Dokumententyp | Veröffentlichungsdatum
Anzahl der Publikationen: 15

Zeitschriftenartikel

Pogoda, K.; Kameritsch, P.; Mannell, H. und Pohl, U. (2019): Connexins in the control of vasomotor function. In: Acta Physiologica, Bd. 225, Nr. 1, UNSP e13108

Kiemer, F.; Kameritsch, P.; Pohl, U. und Pogoda, K. (2017): PKA modulates the migration enhancing effect of Connexin 43 in a negative manner. In: Acta Physiologica, Bd. 219: S. 30

Dendorfer, A.; Fischer, C.; Milting, H.; Fein, E.; Husse, B.; Cao-Ehlker, X. und Pohl, U. (2017): Heart in a dish: A stable in vitro meodel of continuously beating human myocardium built from precision-cut tissue slices and biomimetic cultivation. In: Acta Physiologica, Bd. 219: S. 40

Heun, Y.; Anton, M.; Pfeifer, A.; Pohl, U. und Mannell, H. (2017): SHP-2 promotes hypoxia induced wound healing in vivo by mediating stabilization and activation of HIF-1 alpha. In: Acta Physiologica, Bd. 219: S. 54-56

Qiu, J.; Schubert, K.; Blodow, S.; Wiedenmann, M.; Schneider, H. und Pohl, U. (2017): The AMP-activated protein kinase (AMPK) reduces calcium sensitivity and causes vasodilation by altering actin filament dynamics in microvascular smooth muscle. In: Acta Physiologica, Bd. 219: S. 58

Qiu, J.; Schubert, K. Michael; Schneider, H.; Blodow, S.; Wiedenmann, M. und Pohl, U. (2017): The AMP-Activated Protein Kinase (AMPK) Reduces Calcium Sensitivity And Causes Vasodilation By Altering Actin Filament Dynamics In Microvascular Smooth Muscle. In: Journal of Vascular Research, Bd. 54: S. 23

Heun, Y.; Anton, M.; Pfeifer, A.; Plank, C.; Pohl, U. und Mannell, H. (2017): Lentiviral Magnetic Targeting of SHP-2 Locally Governs HIF-1a Dependent Wound Healing Angiogenesis in vivo. In: Journal of Vascular Research, Bd. 54: S. 37

Fischer, C.; Bruegmann, T.; Fein, E.; Husse, B.; Fey, T.; Sasse, P.; Pohl, U. und Dendorfer, A. (2016): Optogenetic stimulation permits long-term assessment of contractility in organotypic tissue cultures of murine myocardium. In: Acta Physiologica, Bd. 216

Drzisga, D.; Köppl, T.; Pohl, U.; Helmig, R. und Wohlmuth, B. (2016): Numerical modeling of compensation mechanisms for peripheral arterial stenoses. In: Computers in Biology and Medicine, Bd. 70: S. 190-201

Kameritsch, P.; Pogoda, K. und Pohl, U. (2016): The inhibitory effect of NO on Cx37-dependent intercellular calcium signaling targets the phosphorylation state of tyrosine 332. In: Acta Physiologica, Bd. 216

Kiemer, F.; Pohl, U. und Pogoda, K. (2016): PKA dependent phosphorylation of the C-terminal part of Connexin 43 reduces cell migration. In: Acta Physiologica, Bd. 216

Pogoda, K.; Kameritsch, P. und Pohl, U. (2016): Cx43 mediated augmentation of cell motility is associated with enhanced focal adhesion turnover and reduced stress fibres formation. In: Acta Physiologica, Bd. 216

Stampnik, Y.; Kroetz, F.; Pircher, J.; Kretschmann, A.; Pohl, U. und Mannell, H. (2016): The protein-tyrosine phosphatase SHP-2 counteracts IL-1 beta induced endothelial inflammation. In: Acta Physiologica, Bd. 216

Tahir, S.; Wagner, A. H.; Dietzel, S.; Pohl, U. und Hecker, M. (2016): von Willebrand factor mediated platelet string formation and leukocyte extravasation in the microcirculation in vivo. In: Acta Physiologica, Bd. 216

Bolz, S. S. und Pohl, U. (2003): Highly effective non-viral gene transfer into vascular smooth muscle cells of cultured resistance arteries demonstrated by genetic inhibition of sphingosine-1-phosphate-induced vasoconstriction. In: Journal of Vascular Research, Nr. 4: S. 399-405 [PDF, 154kB]

Diese Liste wurde am Sat Mar 23 23:52:56 2024 CET erstellt.