Schmidt, Andreas W. ORCID: https://orcid.org/0000-0001-5609-290X; Kühnapfel, Andreas ORCID: https://orcid.org/0000-0003-3668-0784; Kirsten, Holger ORCID: https://orcid.org/0000-0002-3126-7950; Grallert, Harald ORCID: https://orcid.org/0000-0002-6876-9655; Hellerbrand, Claus; Kiefer, Falk; Mann, Karl; Mueller, Sebastian; Nöthen, Markus M.; Peters, Annette ORCID: https://orcid.org/0000-0001-6645-0985; Ridinger, Monika; Frank, Josef; Rietschel, Marcella; Soranzo, Nicole; Soyka, Michael; Wodarz, Norbert ORCID: https://orcid.org/0000-0001-9070-8448; Malerba, Giovanni; Gambaro, Giovanni ORCID: https://orcid.org/0000-0001-5733-2370; Gieger, Christian; Scholz, Markus; Krug, Sebastian; Michl, Patrick ORCID: https://orcid.org/0000-0002-0401-5600; Ewers, Maren ORCID: https://orcid.org/0000-0002-9029-2240; Witt, Heiko ORCID: https://orcid.org/0000-0001-7791-7341; Laumen, Helmut ORCID: https://orcid.org/0000-0002-5885-5242 und Rosendahl, Jonas ORCID: https://orcid.org/0000-0003-4513-0506
(2022):
Colocalization analysis of pancreas eQTLs with risk loci from alcoholic and novel non-alcoholic chronic pancreatitis GWAS suggests potential disease causing mechanisms.
In: Pancreatology, Bd. 22, Nr. 4: S. 449-456
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