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Eine Ebene höherGruppiert nach: Dokumententyp |
Veröffentlichungsdatum
Anzahl der Publikationen: 7
2022
Krämer, Björn F.; Hennis, Irina; Karge, Anne; Krämer, Anne Katrin; Dreyer, Tobias F.; Kiechle, Marion; Kuschel, Bettina und Bronger, Holger
(2022):
Platelet mitochondrial membrane depolarization reflects disease severity in patients with preeclampsia.
In: Molecular Medicine, Bd. 28, Nr. 1, 51
Krämer, Björn F.; Martinovic, Ivo und Lindemann, Stephan
(2022):
Human platelets release TGFBIp in acute myocardial infarction.
In: Heart and Vessels, Bd. 37, Nr. 11: S. 1962-1970
2019
Krämer, Björn F.; Lamkemeyer, Tobias; Franz-Wachtel, Mirita und Lindemann, Stephan
(2019):
The Integrin Activating Protein Kindlin-3 Is Cleaved in Human Platelets during ST-Elevation Myocardial Infarction.
In: International Journal of Molecular Sciences, Bd. 20, Nr. 24, 6154
Grundler Groterhorst, Katharina; Mannell, Hanna; Pircher, Joachim und Krämer, Björn F.
(2019):
Platelet Proteasome Activity and Metabolism Is Upregulated during Bacterial Sepsis.
In: International Journal of Molecular Sciences, Bd. 20, Nr. 23, 5961
Krämer, Björn F.; Mannell, Hanna; Lamkemeyer, Tobias; Franz-Wachtel, Mirita und Lindemann, Stephan
(2019):
Heat-Shock Protein 27 (HSPB1) Is Upregulated and Phosphorylated in Human Platelets during ST-Elevation Myocardial Infarction.
In: International Journal of Molecular Sciences, Bd. 20, Nr. 23, 5968
Heun, Yvonn; Grundler Groterhorst, Katharina; Pogoda, Kristin; Krämer, Björn F.; Pfeifer, Alexander; Pohl, Ulrich und Mannell, Hanna
(2019):
The Phosphatase SHP-2 Activates HIF-1 alpha in Wounds In Vivo by Inhibition of 26S Proteasome Activity.
In: International Journal of Molecular Sciences, Bd. 20, Nr. 18, 4404
Heun, Yvonn; Pircher, Joachim; Czermak, Thomas; Bluem, Philipp; Hupel, Georg; Bohmer, Monica; Krämer, Björn F.; Pogoda, Kristin; Pfeifer, Alexander; Woernle, Markus; Ribeiro, Andrea; Hübner, Max; Kreth, Simone; Claus, Ralf A.; Weis, Sebastian; Ungelenk, Luisa; Kroetz, Florian; Pohl, Ulrich und Mannell, Hanna
(2019):
Inactivation of the tyrosine phosphatase SHP-2 drives vascular dysfunction in Sepsis.
In: eBioMedicine, Bd. 42: S. 120-132
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