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Anzahl der Publikationen: 6
Zeitschriftenartikel
Feil, Katharina; Remi, Jan; Kuepper, Clemens; Herzberg, Moriz; Dorn, Franziska; Kunz, Wolfgang G.; Rotkopf, Lukas T.; Heinrich, Johanna; Müller, Katharina; Laub, Christoph; Levin, Johannes; Huettemann, Katrin; Dabitz, Rainer; Müller, Robert; Wollenweber, Frank A.; Pfefferkorn, Thomas; Hamann, Gerhard F.; Liebig, Thomas; Dieterich, Marianne und Kellert, Lars
(2020):
Drip and ship for mechanical thrombectomy within the Neurovascular Network of Southwest Bavaria.
In: Neurology, Bd. 94, Nr. 5, E453-E463
Feil, Katharina; Heinrich, Johanna; Küpper, Clemens; Müller, Katharina; Laub, Christoph; Falkenhausen, Aenne S. von; Becker, Regina; Wollenweber, Frank A.; Kääb, Stefan; Sinner, Moritz F. und Kellert, Lars
(2019):
Catch-up-ESUS - follow-up in embolic stroke of undetermined source (ESUS) in a prospective, open-label, observational study: study protocol and initial baseline data.
In: BMJ open, Bd. 9, Nr. 12, e031716
[PDF, 434kB]
Brosel, Sonja; Laub, Christoph; Averdam, Anne; Bender, Andreas und Elstner, Matthias
(2016):
Molecular aging of the mammalian vestibular system.
In: Ageing Research Reviews, Bd. 26: S. 72-80
Patzig, Maximilian; Laub, Christoph; Janssen, Hendrik; Ertl, Lorenz und Fesl, Gunther
(2014):
Pseudo-subarachnoid haemorrhage due to chronic hypoxaemia: case report and review of the literature.
In: BMC Neurology
14:219
[PDF, 728kB]
Bender, Andreas; Desplats, Paula; Spencer, Brian; Rockenstein, Edward; Adame, Anthony; Elstner, Matthias; Laub, Christoph; Mueller, Sarina; Koob, Andrew O.; Mante, Michael; Pham, Emily; Klopstock, Thomas und Masliah, Eliezer
(2013):
TOM40 Mediates Mitochondrial Dysfunction Induced by α-Synuclein Accumulation in Parkinson's Disease.
In: PLOS ONE 8(4), e62277 [PDF, 2MB]
In: PLOS ONE 8(4), e62277 [PDF, 2MB]
Elstner, Matthias; Mueller, Sarina K.; Leidolt, Lars; Laub, Christoph; Krieg, Lena; Schlaudraff, Falk; Liss, Birgit; Morris, Chris; Turnbull, Douglass M.; Masliah, Eliezer; Prokisch, Holger; Klopstock, Thomas und Bender, Andreas
(2011):
Neuromelanin, neurotransmitter status and brainstem location determine the differential vulnerability of catecholaminergic neurons to mitochondrial DNA deletions.
In: Molecular Brain
4:43
[PDF, 5MB]