![[höher]](/style/images/multi_up.png)
Eine Ebene höherGruppiert nach:
Dokumententyp
| Veröffentlichungsdatum
Springe zu: Zeitschriftenartikel
Anzahl der Publikationen: 14
Zeitschriftenartikel
Balbi, M.; Koide, M.; Wellman, G. C. und Plesnila, N.
(2017):
Impairment of neurovascular coupling after subarachnoid hemorrhage in vivo.
In: Journal of Cerebral Blood Flow and Metabolism, Bd. 37: S. 445
Westermayer, I.; Terpolilli, N.; Katzdobler, S. und Plesnila, N.
(2017):
Role of the endothelium NO-Synthase in early brain injury after experimental subarachnoid hemorrhage.
In: Journal of Cerebral Blood Flow and Metabolism, Bd. 37: S. 113
Katzdobler, S.; Seker, B.; Terpolilli, N. A.; Westermayer, I. und Plesnila, N.
(2017):
The sGC-activator Bay 60-2770 has no influence on microvasospasms after experimental subarachnoid hemorrhage.
In: Journal of Cerebral Blood Flow and Metabolism, Bd. 37: S. 118-119
Liesz, Arthur; Planas, A. M.; De Simoni, M. G.; Plesnila, N.; Vivien, D. und Dirnagl, U.
(2016):
Results of the first preclinical multicenter trial: Anti-CD49d treatment in acute brain ischemia.
In: Journal of Cerebral Blood Flow and Metabolism, Bd. 36, Nr. Suppl. 1: S. 132-133
Schwarzmaier, S. M.; de Chaumont, C.; Balbi, M.; Terpolilli, N. A.; Kleinschnitz, C.; Gruber, A. und Plesnila, N.
(2016):
Evaluation of microthrombosis in the parenchymal circulation after experimental traumatic brain injury by in vivo imaging - role of factor XI.
In: Journal of Cerebral Blood Flow and Metabolism, Bd. 36: S. 298
Balbi, M. und Plesnila, N.
(2016):
Neurovascular coupling after subarachnoid hemorrhage (SAH) in vivo.
In: Journal of Cerebral Blood Flow and Metabolism, Bd. 36: S. 343
Bühler, D.; Azghandi, S.; Schüller, K. und Plesnila, N.
(2016):
Decompressive craniectomy after subarachnoid hemorrhage - experimental results in mice.
In: Journal of Cerebral Blood Flow and Metabolism, Bd. 36: S. 345
Terpolilli, N.; Dienel, A.; Heumos, N.; Schoeller, K. und Plesnila, N.
(2016):
Inhibition of endothelin-1-receptors (et-a-receptor) does not influence microvasospasm and neurological outcome after experimental subarachnoid hemorrhage.
In: Journal of Cerebral Blood Flow and Metabolism, Bd. 36: S. 364-365
Azghandi, S.; Bühler, D.; Mamrak, U.; Schneider, M.; Dichgans, Martin 
ORCID: https://orcid.org/0000-0002-0654-387X und Plesnila, N.
(2016):
Effect of HDAC9 deficiency on ischemic stroke outcome.
In: Journal of Cerebral Blood Flow and Metabolism, Bd. 36: S. 485-486
ORCID: https://orcid.org/0000-0002-0654-387X und Plesnila, N.
(2016):
Effect of HDAC9 deficiency on ischemic stroke outcome.
In: Journal of Cerebral Blood Flow and Metabolism, Bd. 36: S. 485-486
Terpolilli, N.; Dienel, A.; Schwarzmaier, S. und Plesnila, N.
(2016):
Hypercapnia-induced cerebral vasodilation is mediated predominantly by ENOS.
In: Journal of Cerebral Blood Flow and Metabolism, Bd. 36: S. 543
Balbi, M.; Hanecker, P.; Dichgans, Martin ORCID: https://orcid.org/0000-0002-0654-387X und Plesnila, N.
(2016):
Characterization of cerebrovascular function in mice with cerebral small vessel disease.
In: Journal of Cerebral Blood Flow and Metabolism, Bd. 36: S. 679-680
ORCID: https://orcid.org/0000-0002-0654-387X und Plesnila, N.
(2016):
Characterization of cerebrovascular function in mice with cerebral small vessel disease.
In: Journal of Cerebral Blood Flow and Metabolism, Bd. 36: S. 679-680
Balbi, M.; Ghosh, M.; Longden, T. A.; Jativa, M.; Gesierich, B.; Hellal, F.; Lourbopoulos, A.; Nelson, M. T. und Plesnila, N.
(2016):
Dysfunction of mouse cerebral arteries during early aging.
In: Journal of Cerebral Blood Flow and Metabolism, Bd. 36: S. 174
Dolga, A. M.; de Andrade, A.; Meissner, L.; Knaus, H-G; Hoellerhage, M.; Christophersen, P.; Zischka, H.; Plesnila, N.; Hoeglinger, G. U. und Culmsee, C.
(2014):
Subcellular expression and neuroprotective effects of SK channels in human dopaminergic neurons.
In: Cell Death & Disease, Bd. 5, e999
Doti, N.; Reuther, C.; Scognamiglio, P. L.; Dolga, A. M.; Plesnila, N.; Ruvo, M. und Culmsee, C.
(2014):
Inhibition of the AIF/CypA complex protects against intrinsic death pathways induced by oxidative stress.
In: Cell Death & Disease, Bd. 5, e993