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Anzahl der Publikationen: 15
Zeitschriftenartikel
Pogoda, K.; Kameritsch, P.; Mannell, H. und Pohl, U.
(2019):
Connexins in the control of vasomotor function.
In: Acta Physiologica, Bd. 225, Nr. 1, UNSP e13108
Kiemer, F.; Kameritsch, P.; Pohl, U. und Pogoda, K.
(2017):
PKA modulates the migration enhancing effect of Connexin 43 in a negative manner.
In: Acta Physiologica, Bd. 219: S. 30
Dendorfer, A.; Fischer, C.; Milting, H.; Fein, E.; Husse, B.; Cao-Ehlker, X. und Pohl, U.
(2017):
Heart in a dish: A stable in vitro meodel of continuously beating human myocardium built from precision-cut tissue slices and biomimetic cultivation.
In: Acta Physiologica, Bd. 219: S. 40
Heun, Y.; Anton, M.; Pfeifer, A.; Pohl, U. und Mannell, H.
(2017):
SHP-2 promotes hypoxia induced wound healing in vivo by mediating stabilization and activation of HIF-1 alpha.
In: Acta Physiologica, Bd. 219: S. 54-56
Qiu, J.; Schubert, K.; Blodow, S.; Wiedenmann, M.; Schneider, H. und Pohl, U.
(2017):
The AMP-activated protein kinase (AMPK) reduces calcium sensitivity and causes vasodilation by altering actin filament dynamics in microvascular smooth muscle.
In: Acta Physiologica, Bd. 219: S. 58
Qiu, J.; Schubert, K. Michael; Schneider, H.; Blodow, S.; Wiedenmann, M. und Pohl, U.
(2017):
The AMP-Activated Protein Kinase (AMPK) Reduces Calcium Sensitivity And Causes Vasodilation By Altering Actin Filament Dynamics In Microvascular Smooth Muscle.
In: Journal of Vascular Research, Bd. 54: S. 23
Heun, Y.; Anton, M.; Pfeifer, A.; Plank, C.; Pohl, U. und Mannell, H.
(2017):
Lentiviral Magnetic Targeting of SHP-2 Locally Governs HIF-1a Dependent Wound Healing Angiogenesis in vivo.
In: Journal of Vascular Research, Bd. 54: S. 37
Fischer, C.; Bruegmann, T.; Fein, E.; Husse, B.; Fey, T.; Sasse, P.; Pohl, U. und Dendorfer, A.
(2016):
Optogenetic stimulation permits long-term assessment of contractility in organotypic tissue cultures of murine myocardium.
In: Acta Physiologica, Bd. 216
Drzisga, D.; Köppl, T.; Pohl, U.; Helmig, R. und Wohlmuth, B.
(2016):
Numerical modeling of compensation mechanisms for peripheral arterial stenoses.
In: Computers in Biology and Medicine, Bd. 70: S. 190-201
Kameritsch, P.; Pogoda, K. und Pohl, U.
(2016):
The inhibitory effect of NO on Cx37-dependent intercellular calcium signaling targets the phosphorylation state of tyrosine 332.
In: Acta Physiologica, Bd. 216
Kiemer, F.; Pohl, U. und Pogoda, K.
(2016):
PKA dependent phosphorylation of the C-terminal part of Connexin 43 reduces cell migration.
In: Acta Physiologica, Bd. 216
Pogoda, K.; Kameritsch, P. und Pohl, U.
(2016):
Cx43 mediated augmentation of cell motility is associated with enhanced focal adhesion turnover and reduced stress fibres formation.
In: Acta Physiologica, Bd. 216
Stampnik, Y.; Kroetz, F.; Pircher, J.; Kretschmann, A.; Pohl, U. und Mannell, H.
(2016):
The protein-tyrosine phosphatase SHP-2 counteracts IL-1 beta induced endothelial inflammation.
In: Acta Physiologica, Bd. 216
Tahir, S.; Wagner, A. H.; Dietzel, S.; Pohl, U. und Hecker, M.
(2016):
von Willebrand factor mediated platelet string formation and leukocyte extravasation in the microcirculation in vivo.
In: Acta Physiologica, Bd. 216
Bolz, S. S. und Pohl, U.
(2003):
Highly effective non-viral gene transfer into vascular smooth muscle cells of cultured resistance arteries demonstrated by genetic inhibition of sphingosine-1-phosphate-induced vasoconstriction.
In: Journal of Vascular Research, Nr. 4: S. 399-405
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