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Antoniolli, Martina 
ORCID: https://orcid.org/0000-0003-2720-9886; Solovey, Maria; Hildebrand, Johannes Adrian ORCID: https://orcid.org/0000-0002-5451-8038; Freyholdt, Tabea; Strobl, Carolin Dorothea; Bararia, Deepak; Keay, William David; Adolph, Louisa ORCID: https://orcid.org/0009-0006-4308-2423; Heide, Michael; Passerini, Verena; Winter, Lis; Wange, Lucas ORCID: https://orcid.org/0000-0002-3275-9156; Enard, Wolfgang ORCID: https://orcid.org/0000-0002-4056-0550; Thieme, Susanne; Blum, Helmut ORCID: https://orcid.org/0000-0002-3994-2332; Rudelius, Martina ORCID: https://orcid.org/0000-0001-6928-9955; Mergner, Julia ORCID: https://orcid.org/0000-0002-4332-1280; Ludwig, Christina; Bultmann, Sebastian; Schmidt-Supprian, Marc ORCID: https://orcid.org/0000-0002-8543-6166; Leonhardt, Heinrich ORCID: https://orcid.org/0000-0002-5086-6449; Subklewe, Marion ORCID: https://orcid.org/0000-0001-9154-9469; von Bergwelt-Baildon, Michael; Colomé-Tatché, Maria ORCID: https://orcid.org/0000-0002-2224-7560 und Weigert, Oliver ORCID: https://orcid.org/0000-0002-0987-7373
(2025):
ARID1A mutations protect follicular lymphoma from FAS-dependent immune surveillance by reducing RUNX3/ETS1-driven FAS-expression.
In: Cell Death & Differentiation, Bd. 32, Nr. 5: S. 899-910
[PDF, 4MB]
ORCID: https://orcid.org/0000-0003-2720-9886; Solovey, Maria; Hildebrand, Johannes Adrian ORCID: https://orcid.org/0000-0002-5451-8038; Freyholdt, Tabea; Strobl, Carolin Dorothea; Bararia, Deepak; Keay, William David; Adolph, Louisa ORCID: https://orcid.org/0009-0006-4308-2423; Heide, Michael; Passerini, Verena; Winter, Lis; Wange, Lucas ORCID: https://orcid.org/0000-0002-3275-9156; Enard, Wolfgang ORCID: https://orcid.org/0000-0002-4056-0550; Thieme, Susanne; Blum, Helmut ORCID: https://orcid.org/0000-0002-3994-2332; Rudelius, Martina ORCID: https://orcid.org/0000-0001-6928-9955; Mergner, Julia ORCID: https://orcid.org/0000-0002-4332-1280; Ludwig, Christina; Bultmann, Sebastian; Schmidt-Supprian, Marc ORCID: https://orcid.org/0000-0002-8543-6166; Leonhardt, Heinrich ORCID: https://orcid.org/0000-0002-5086-6449; Subklewe, Marion ORCID: https://orcid.org/0000-0001-9154-9469; von Bergwelt-Baildon, Michael; Colomé-Tatché, Maria ORCID: https://orcid.org/0000-0002-2224-7560 und Weigert, Oliver ORCID: https://orcid.org/0000-0002-0987-7373
(2025):
ARID1A mutations protect follicular lymphoma from FAS-dependent immune surveillance by reducing RUNX3/ETS1-driven FAS-expression.
In: Cell Death & Differentiation, Bd. 32, Nr. 5: S. 899-910
[PDF, 4MB]
Bortoluzzi, Sabrina; Dashtsoodol, Nyambayar; Engleitner, Thomas; Drees, Christoph; Helmrath, Sabine; Mir, Jonas; Toska, Albulena; Flossdorf, Michael; Oellinger, Rupert; Solovey, Maria; Colome-Tatche, Maria; Kalfaoglu, Bahire; Ono, Masahiro; Buch, Thorsten; Ammon, Tim; Rad, Roland und Schmidt-Supprian, Marc
(2021):
Brief homogeneous TCR signals instruct common iNKT progenitors whose effector diversification is characterized by subsequent cytokine signaling.
In: Immunity, Bd. 54, Nr. 11: S. 2497-2513
Hecker, Judith S.; Hartmann, Luise; Riviere, Jennifer; Buck, Michele C.; Garde, Mark van de; Rothenberg-Thurley, Maja; Fischer, Luise; Winter, Susann; Ksienzyk, Bianka; Ziemann, Frank; Solovey, Maria; Rauner, Martina; Tsourdi, Elena; Sockel, Katja; Schneider, Marie; Kubasch, Anne S.; Nolde, Martin; Hausmann, Dominikus; Paulus, Alexander C.; Luetzner, Joerg; Roth, Andreas; Bassermann, Florian; Spiekermann, Karsten; Marr, Carsten; Hofbauer, Lorenz C.; Platzbecker, Uwe; Metzeler, Klaus H. und Goetze, Katharina S.
(2021):
CHIP and hips: clonal hematopoiesis is common in patients undergoing hip arthroplasty and is associated with autoimmune disease.
In: Blood, Bd. 138, Nr. 18: S. 1727-1732
Solovey, Maria; Wang, Ying; Michel, Christian; Metzeler, Klaus H.; Herold, Tobias; Goethert, Joachim R.; Ellenrieder, Volker; Hessmann, Elisabeth; Gattenloehner, Stefan; Neubauer, Andreas; Pavlinic, Dinko; Benes, Vladimir; Rupp, Oliver und Burchert, Andreas
(2019):
Nuclear factor of activated T-cells, NFATC1, governs FLT3(ITD)-driven hematopoietic stem cell transformation and a poor prognosis in AML.
In: Journal of Hematology & Oncology, Bd. 12, 72