Anzahl der Publikationen: 3
	Zeitschriftenartikel
    Satoh, Takashi K.  ORCID: https://orcid.org/0000-0002-6064-9253; Neulinger, Matthias Munoz
ORCID: https://orcid.org/0000-0002-6064-9253; Neulinger, Matthias Munoz  ORCID: https://orcid.org/0000-0001-5406-0402; Stadler, Pia‐Charlotte
ORCID: https://orcid.org/0000-0001-5406-0402; Stadler, Pia‐Charlotte  ORCID: https://orcid.org/0000-0001-5892-4925; Aoki, Rui
ORCID: https://orcid.org/0000-0001-5892-4925; Aoki, Rui  ORCID: https://orcid.org/0000-0001-7061-4508 und French, Lars E.
ORCID: https://orcid.org/0000-0001-7061-4508 und French, Lars E.  ORCID: https://orcid.org/0000-0002-4629-1486
  
(2024):
		Immune checkpoint inhibitor‐induced epidermal necrolysis: A narrative review evaluating demographics, clinical features, and culprit medications.
	
	 In: The Journal of Dermatology, Bd. 51, Nr.  1: S. 3-11
	
      
        
          
             [PDF, 5MB]
ORCID: https://orcid.org/0000-0002-4629-1486
  
(2024):
		Immune checkpoint inhibitor‐induced epidermal necrolysis: A narrative review evaluating demographics, clinical features, and culprit medications.
	
	 In: The Journal of Dermatology, Bd. 51, Nr.  1: S. 3-11
	
      
        
          
             [PDF, 5MB]
          
        
      
 
    Calabrese, Laura  ORCID: https://orcid.org/0000-0001-5238-2336; Ney, Francesca; Aoki, Rui
ORCID: https://orcid.org/0000-0001-5238-2336; Ney, Francesca; Aoki, Rui  ORCID: https://orcid.org/0000-0001-7061-4508; Moltrasio, Chiara; Marzano, Angelo V.; Kerl, Katrin; Stadler, Pia‐Charlotte; Satoh, Takashi K. und French, Lars E.
  
(2023):
		Characterisation of IL‐1 family members in Sweet syndrome highlights the overexpression of IL‐1β and IL‐1R3 as possible therapeutic targets.
	
	 In: Experimental Dermatology, Bd. 32, Nr.  11: S. 1915-1923
	
      
        
          
             [PDF, 2MB]
ORCID: https://orcid.org/0000-0001-7061-4508; Moltrasio, Chiara; Marzano, Angelo V.; Kerl, Katrin; Stadler, Pia‐Charlotte; Satoh, Takashi K. und French, Lars E.
  
(2023):
		Characterisation of IL‐1 family members in Sweet syndrome highlights the overexpression of IL‐1β and IL‐1R3 as possible therapeutic targets.
	
	 In: Experimental Dermatology, Bd. 32, Nr.  11: S. 1915-1923
	
      
        
          
             [PDF, 2MB]
          
        
      
 
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