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Eine Ebene höherGruppiert nach: Dokumententyp |
Veröffentlichungsdatum
Anzahl der Publikationen: 5
2019
Luque-Martin, Rosario; Van den Bossche, Jan; Furze, Rebecca C.; Neele, Annette E.; Velden, Saskia van der; Gijbels, Marion J. J.; Roomen, Cindy P. P. A. van; Bernard, Sharon G.; Jonge, Wouter J. de; Rioja, Inmaculada; Prinjha, Rab K.; Lewis, Huw D.; Mander, Palwinder K. und Winther, Menno P. J. de
(29. Oktober 2019):
Targeting Histone Deacetylases in Myeloid Cells Inhibits Their Maturation and Inflammatory Function With Limited Effects on Atherosclerosis.
In: Frontiers in Pharmacology, Bd. 10, 1242
[PDF, 4MB]
Ketelhuth, Daniel F. J.; Lutgens, Esther; Back, Magnus; Binder, Christoph J.; Van den Bossche, Jan; Daniel, Carolin; Dumitriu, Ingrid E.; Hoefer, Imo; Libby, Peter; O'Neill, Luke; Weber, Christian und Evans, Paul C.
(2019):
Immunometabolism and atherosclerosis: perspectives and clinical significance: a position paper from the Working Group on Atherosclerosis and Vascular Biology of the European Society of Cardiology.
In: Cardiovascular Research, Bd. 115, Nr. 9: S. 1385-1392
Wenstedt, Eliane F. E.; Verberk, Sanne G. S.; Kroon, Jeffrey; Neele, Annette E.; Baardman, Jeroen; Claessen, Nike; Pasaoglu, Ozge T.; Rademaker, Emma; Schrooten, Esmee M.; Wouda, Rosa D.; de Winther, Menno P. J.; Aten, Jan; Vogt, Liffert und Van den Bossche, Jan
(2019):
Salt increases monocyte CCR2 expression and inflammatory responses in humans.
In: Jci Insight, Bd. 4, Nr. 21, e130508
van Beek, Adriaan A.; Van den Bossche, Jan; Mastroberardino, Pier G.; de Winther, Menno P. J. und Leenen, Pieter J. M.
(2019):
Metabolic Alterations in Aging Macrophages: Ingredients for Inflammaging?
In: Trends in Immunology, Bd. 40, Nr. 2: S. 113-127
2016
Van den Bossche, Jan; Baardman, Jeroen; Otto, Natasja A.; Velden, Saskia van der; Neele, Annette E.; Berg, Susan M. van den; Luque-Martin, Rosario; Chen, Hung-Jen; Boshuizen, Marieke C. S.; Ahmed, Mohamed; Hoeksema, Marten A.; de Vos, Alex F. und de Winther, Menno P. J.
(2016):
Mitochondrial Dysfunction Prevents Repolarization of Inflammatory Macrophages.
In: Cell Reports, Bd. 17, Nr. 3: S. 684-696
[PDF, 4MB]